Professor Rob Hegele discusses the complex and not yet fully understood genetic basis of pancreatitis, including rare gain-of-function mutations that make pathological conversion from trypsinogen to trypsin in the pancreas more likely, as well as rare loss-of-function mutations in conversion inhibitors which also result in inappropriate trypsin production.
Educational partners and supporters
Financial support was provided as a GMG Knowledge Gap Grant from Pfizer Inc.
Financial support was provided as a GMG Knowledge Gap Grant from Pfizer Inc.
Financial support was provided as a GMG Knowledge Gap Grant from Pfizer Inc.
